Plantar fasciitis is a degenerative condition of the plantar fascia — the thick fibrous band running from the calcaneal tubercle to the bases of the proximal phalanges — not an inflammatory one, a distinction with the same clinical importance as the tendinitis versus tendinopathy distinction in Achilles disease. The "itis" terminology has persisted despite histological studies consistently showing degenerative change, disorganized collagen, and fibroblast proliferation rather than acute inflammatory infiltrate. This matters because anti-inflammatory treatments (NSAIDs, cortisone) address a process that is not the primary pathology.
Why the First Steps of the Morning Hurt the Most
The cardinal symptom of plantar fasciitis — severe pain in the heel with the first steps after a period of rest — has a mechanical explanation. During sleep, the ankle is in a relatively plantarflexed position. This shortens the plantar fascia. The first loading of the foot in the morning suddenly stretches a shortened, stiff, structurally compromised structure, producing the sharp pain that patients describe as "stepping on glass." The pain often improves after 10–20 minutes of walking because the fascia warms and becomes more extensible under repetitive low-load stretching — and then returns with sustained activity as cumulative load accumulates.
The High-Load Heel Raise Protocol
Roos and colleagues published a landmark randomized controlled trial in 2006 demonstrating that a high-load strengthening exercise — a single-leg heel raise on an incline with the toes in extension — produced superior outcomes to plantar-specific stretching alone. The proposed mechanism: the combined loading of the plantar fascia and Achilles tendon complex during an inclined heel raise, with toe extension adding passive tension through the windlass mechanism, provides the mechanical stimulus necessary for collagen remodeling in a degenerative fascia.
Where Cortisone Fits — and Where It Does Not
Cortisone injection to the plantar fascia insertion produces reliable short-term pain relief at 4–8 weeks. It does not accelerate the underlying collagen remodeling and has no benefit over loading programs at 6–12 months in controlled trials. The risk worth knowing: repeated cortisone injections to the plantar fascia (more than two to three total) significantly increase the risk of plantar fascia rupture and fat pad atrophy. A single injection to control acute-phase pain while initiating a loading program is a reasonable strategy. Using cortisone as the primary treatment, repeated at every flare, is not.
Extracorporeal shockwave therapy (ESWT) has reasonable evidence for plantar fasciitis refractory to 3–6 months of conservative care, and is preferred over surgery in most cases. Surgical plantar fasciotomy — partial or complete release — is reserved for a small minority of patients who fail all conservative and minimally invasive measures over 12 months.
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